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Minelli C, Kikuta A, Tsud N, Ball MD, Yamamoto A. A microfluidic study of whole blood behaviour on PMMA topographical nanostructures. J Nanobiotechnology. 2008;6:3. 3. Orr MW, Boullin DJ. The relationship between changes in 5-HT induced platelet aggregation and clinical state in patients treated with Fluphenazine. Br J Clin Pharmacol. 1976;3(5):925-8. 4. Rendu F, Brohard-Bohn B. The platelet release reaction: granules’ constituents, secretion and functions. Platelets. 2001;12(5):261-73. 5. Holmsen H.

3. Lokeshwar MR, Dalal R, Manglani M, Shah N. Anemia in newborn. Ind J Pediatr. 1998;65:651-61. 4. Usher R, Shepard M, Lind J. The blood volume of newborn infant and placental transfusion. Acta Paediatric Scand. 1963;52:497-512. 5. Mollison PL, Veall N, Cutbush M. Red cell and plasma volume in newborn infants. Arch Dis Child. 1950;25:242-53. 6. Dochain J, Lemage L, Lambrechts A. Cited in Oski and Naiman. Normal blood values in newborn period. Hematologic problems in the newborn. WB Saunders Co, Philadelphia.

3-7 With mild stimulus, dense granule products are released and with stronger stimulus alpha granules and finally acid hydrolases from lysosome are released. Further Aggregation and Activation of Coagulation As the reinforcement of adhesion and aggregation conti­ nues at the site to tissue injury, the liquid phase of blood B D E Figs 3A to E Platelet aggregometry (A) Born principle, (B) ADP-induced platelet aggregation, (C) Collagen-induced platelet aggregation, (D) Ristocetin-induced platelet aggregation, and (E) Arachidonic aggregation acid-induced platelet aggregation 18 Section-1 Physiology coagulation is also activated.

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Textbook of Pediatric Hematology & Hemato-Oncology by coll.

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